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The authors concluded that the findings of sudden cardiac deaths attributable to COVID vaccine-related myocarditis warrant the careful monitoring or warning of sudden cardiac death as a potentially fatal complication of COVID-19 vaccination, especially in individuals younger than 45 years of age who received the mRNA vaccines.
The S1 protein activates human CFs by priming NLRP3 inflammasomes through NF-κB signaling in an ACE2-dependent way.
This study showed for the first time that mRNA-1273 and BNT162b2 COVID-19 vaccines induced cardiotoxic effects with disturbances of normal contractile function in rat cardiomyocytes. The effects of vaccines differed fundamentally in their pathophysiological mechanisms.
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Individuals who were asymptomatic and underwent PET/CT within a period of 180 days following their second vaccination had increased 18Fluorine-fluorodeoxyglucose uptake in the myocardium, axilla, liver, and spleen as compared to unvaccinated individuals.
In this retrospective pharmacovigilance study of safety signals associated with myocarditis/pericarditis after the primary and up to three booster doses of mRNA COVID-19 vaccines, the second dose of the vaccines showed the strongest signal in vaccinated individuals under 25 years old.
30% of patients diagnosed with long COVID syndrome had cardiac MRI signs of non-ischemic fibrosis, exceeding the prevalence of this disease in the normal adult population.
PET and MRI visualization of myocardial tissue has shown localized myocardial inflammation and edema in a small proportion of patients with symptomatic myocarditis approximately two months after COVID-19 vaccination.Â
The results showed the possible association between anti-Dsg2 autoantibodies and post-COVID-19 cardiac sequelae.Â
